Bill Hartman 12:10–15:02
Ryan starts out with, how do you determine if a proxy measure of the extremity is pathological? And so he's referring to ligamentous laxity or capsular instability. On the opposite end, how would you determine a true tissue extensibility limitation assuming you've maximized axial position and respiratory variability? How would you treat these two presentations differently? So let's attack this from the beginning. So how do you determine proxy measures in the extremity pathological? Well, first and foremost, the history is going to give you a lot of information in that regard. So if somebody has a dislocation episode of some sort, then chances are you're going to be dealing with some form of tissue adaptation or a traumatic instability. And so again, when you have a history like that, we're going to make an assumption that we do have a pathology. I think a lot of people, and I think this is where your question is going, is that when we get a measure that seems to be abnormal in regard to a larger excursion than expected, a lot of people will jump on the bandwagon that, oh, we have ligamentous laxity. The thing that you have to consider under those situations is if we have an orientation of the thorax or an orientation of the pelvis, we can get a magnification of what we perceive as to be the normal excursion range of motion. So let me give you for instance. So if I can anteriorly orient a pelvis enough that the acetabulum is facing more downward, I can get an excessive amount of total excursion of hip range of motion. So under normal circumstances, we might say that extra rotation is 60 degrees from the imaginary zero point and 40 degrees of internal rotation from the imaginary zero point, so that's 100 degrees of excursion. But if I can actually orient that pelvis enough to get the anteverted acetabular position that allows it, I might even capture 120 to 130 degrees of total hip excursion, but that's just an orientation problem. That is not indicative of the fact that somebody has developed some imaginary capsular laxity, so don't immediately rush to judgment under those circumstances. Secondarily, I don't know how much that changes things a whole lot anyway. The goal is to acquire dynamic control of orientation of the axial skeleton and dynamic control of the excursion of the peripheral joints, and that's a muscular control issue rather than just looking at the laxity issues. Granted, we might have some proprioceptive deficits that are associated with a true laxity or some form of traumatic laxity that occurs, but under most circumstances, I think that's going to be determined through process. So when I have somebody that can't consistently recapture a position or can't hold on to their changes, now you may have a situation where we have a structural adaptation that is influencing the outcome and that might be when we have to make the consult with the orthopedic surgeon under those circumstances. But that's how you're going to determine this, Ryan. It's going to be part of the process. It's not about making an immediate leap because people overcome things all the time. People walk in with rotator cuff tears that they will overcome; those people come in with these perceived laxities and they're able to overcome those. So again, I always default to you make the attempts first; you run yourself through the process and you see what they can reacquire; you see what they get they can learn to control. And if that doesn't work, that's when you start to make the assumption that, okay, maybe I do have a situation where structure has changed to such a degree and that I need to bring in another element of service or integration. So hopefully that answers that first part. When we talk about a tissue, would you call it a tissue extensibility limitation? So the thing that pops into my head under those circumstances or situations where maybe we have a person that comes in that has been diagnosed with a frozen shoulder. And so under those circumstances, what would happen as you run through the process as you attempt your global or more systemic influences in treatment? You'll see that you don't get the local changes that you expect. In that circumstance, now you need to think about what you have in your toolbox that will address those local issues. Maybe you do have a tissue adaptation that can occur under those circumstances. And so again, that's going to be just a longer process, and so your strategies will be a little bit different because if you're truly looking at a tissue adaptation, it's rare; I think those situations actually occur. I think there's a lot of other influences that are going on in regard to the way that the nervous system is behaving and based on the way that those changes take place because some of those adaptations, if they were true, I don't think all of those would be recapturable, especially with some of the capsular adaptations and then the aggression with which you would have to apply forces to make those changes. Having worked with a fair amount of people that have been diagnosed with frozen shoulders and seeing what is truly able to be recaptured, I don't think that we're looking at those adaptations necessarily. I think we're looking at a lot of influences of the nervous system which can occur locally. We do have local environmental changes in regard to the shape change of the proteins themselves. So we talk about the contractual elements changing. And so those environments can change with different forms of manual therapy, different influences of medication, etc. So again, I don't think they occur as much as many people blame them on. I think that we just need to look at expanding our perspective in our toolbox. But under those circumstances, if you do have a true tissue limitation, then maybe you're looking at a very, very long-term strategy in regards to trying to add length to tissues. And that would be using some of your dinosaur plant strategies, perhaps, a lot of time and effort you applied by the client to restore that range of motion through repetition and avoiding situations where you're creating a negative influence associated with pain and discomfort and in ranges and just reacquiring some of that range of motion through the desensitization strategies. So I think there's many ways to go about this, but again, I caution you to make against making that leap that you're making an assumption that you do have a tissue change. They do most likely occur; I just don't think they occur as often as we think. The second half of Ryan's question piggybacks off of a question from last week where you ask what tests or measures do you apply to determine whether the elbows are oriented towards ER pronation or IR supination. Ryan, this is actually pretty straightforward because assuming we're doing table tests and such, we would have our axial representation. We would have extremity measures that we would have taken that would tell us whether we have a humeral position in external rotation or internal rotation, again, based on those table tests. And then it's just a matter of looking at the pronation and supination available at the elbow. And so if we have identified a shoulder that is in humeral external rotation, we would stabilize the epicondyle at the elbow, we would check our pronation supination and come up with a determination what we're looking at in the forearm. However, I would offer you this that you probably need to look at the wrist for confirmation of that forearm position because in some cases you may have eccentric orientation at the elbow or concentric orientation at the elbow that will skew your perspective. So if we add the wrist measures into this, so we've got a shoulder, we've got an elbow, and then we've got the wrist measures, if you have a deficit in ulna deviation and extension, you most likely have a pronated forearm. And if you have a deviation inflection radial deviation, then you probably have a supinated form. And that's just the orientation of the distal radial ulnar position. When the form is pronated, there is this perceived retraction of the radius that would position it more towards radial deviation and the ulna would appear long under those circumstances, so we would lose that ulna deviation. So that's what occurs in pronation as the radius crosses over the ulna. So just being aware of that, now you have your shoulder elbow and wrist providing you that information to give you the determination of what position and orientation you're looking at at the elbow and then it's just a matter of, do I need a retraction strategy? Do I need an inhalation strategy? And then what orientation do I need to put the forearm and humerus into to restore the ability to capture that full excursion at the elbow? So Ryan has a third part to his question. It goes as such: from a practical standpoint, what do we do with a narrow Campari angle versus a wide Campari angle? I understand that is a representation of the superficial helical angle that compresses the underlying axial helices. But how does it actually change our approach to gaining more variability or more performance? So the Campari angle is the angle formed by the clavicle and the scapula with its apex at the midpoint of the acromion. And so what this actually does measure is how much of a compressive strategy that we have with the superficial musculature. So it's not representative of the superficial helical angle as much as it is representative of how much of a compression strategy we have. So if we have superficial musculature that is compressing the axial skeleton, what happens is the scapula will begin to elevate, and so we get this compression between the anterior and posterior aspect of clavicle scapula and so it compresses that angle and it rides up on the thorax. And so that's why we get this narrowing of the angle. So a normal angle is about 60 degrees give or take. And so if we see that we have a compressed angle there, then we know we have a much more superficial compressive strategy. So from an approach standpoint, there are any number of ways that we can actually reduce that compressive strategy. So under many circumstances, we may just be able to reorient the movement system such that we reduce the compressive strategy and we would see an expansion of that Campari angle. There's a video on YouTube that I talked about earlier that shows a manual technique to actually reduce that compressive strategy. In the gym, we may use something like a suitcase carry that actually reduces that strategy. And so again, there's any number of ways, but as far as using that measurement, it is a useful measurement; it's not an absolute by any stretch of the imagination. Obviously because we named it after one of the padawans, I'm just giving them a hard time. But again, what I don't want you to do is think that it changes a lot in regard to what strategy you may be using.
ligamentous laxity assessmenttissue extensibility limitationsextremity orientation testingCampari angle measurementperipheral joint excursion