Bill Hartman 40:49–43:26
So when we look at the structure of the synovial joint, on either end, as long as we maintain our hyaline cartilage intact, it appears that we can keep our bones apart. So we have to look at what affects that hyaline cartilage, and we say, oh, pressure, tension, blah, blah, blah, blah. But the reality is, Highland Cartilage gets its nutrition from the bony side. So you'll see the little arteries that I drew on my picture here. And that blood supply is what gives the nutrition to the cartilage. So it diffuses from the bloodstream towards the Highland Cartilage on the bony side. Well, if I put enough pressure and tension on those bones, those trabecula will compress. If the trabecula at the ends of the bone compress enough, I restrict the blood flow to the ends of the joint. Now, these trabecula can also fracture. So, you know, you play 15 years in the NBA, you're probably going to get some fracturing of those trabecula. They're kind of like shock absorbers. If you've ever driven on the on the interstate and you see the trash barrels right below the abutment of the overpass and what those are, they're trash barrels filled with water so if you drive off the road and you hit them it'll slow you down so you don't slam right into the bridge. Tribecula kind of the same way they're kind of like shock absorbers so they're filled with with space and water and so when you land they compress but they can fracture over time and then you compress and then the subconvial bone actually gets denser and so you'll see this in arthritic research well this they'll see the the precipitating changes of the So condor bone gets denser and denser and denser. Well that's gonna reduce our blood flow to the cartilage. The cartilage will slowly wear away and it gets thinner and thinner and thinner. So now we're losing our electromagnetic effect. So now we can't keep the joint farther and farther apart. And so now we do get compressive strategies that will actually become destructive. And so again, on that end, that's pretty much how I see a lot of these arthritic changes occurring because it's a pressure-related phenomenon. It's a blood flow-related phenomenon. nutrition to the cartilage. By the way, discs do the same thing. Okay, don't tell anybody. Now, how do we get medial compartment versus lateral compartment? So now we've got to think about our propulsive strategies. So our propulsive strategies are what we apply into the ground. And so propulsion in and of itself is biased towards internal rotation. So we have to apply pressure into the ground. So remember when, when we evolved, we were, we were actually rotated. We were swimmers. We came up on land. We had to learn how to internally rotate and press into the ground. Johnny, when we talk about the internal rotation, I got to internally rotate my femur. because I got to drive down into the ground through internal rotation. So more often than not, I'm going to be applying a little bit more force towards that medial compartment as I internally rotate the femur to push into the ground. And so if we talk about the pressure mechanism that we just talked about in regard to the arthritis, that's why we would probably see the bias towards more medial compartment problems than lateral compartment problems because we're applying forces into the ground. We have to just because of gravity. Okay, so I'm going to breathe for a second. That's a lot to cover. Hope you guys have some questions. I'd be happy to answer those to the best of my ability. But that's kind of what we're talking about. Bones not touching and how we develop arthritis in a nutshell. I hope it was useful for you. Have a great weekend and I'll see you next week.
osteoarthritishyaline cartilagesynovial jointpropulsive strategiesmedial vs lateral compartment